cell nuclear antigen inhibitor Search Results


93
Rockland Immunochemicals rabbit anti pdcd4
Rabbit Anti Pdcd4, supplied by Rockland Immunochemicals, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/cell+nuclear+antigen+inhibitor/pmc03347423-202-41-43?v=Rockland+Immunochemicals
Average 93 stars, based on 1 article reviews
rabbit anti pdcd4 - by Bioz Stars, 2026-07
93/100 stars
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90
ProSci Incorporated pdcd4
Pdcd4, supplied by ProSci Incorporated, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/cell+nuclear+antigen+inhibitor/pmc02907287-110-31-32?v=ProSci+Incorporated
Average 90 stars, based on 1 article reviews
pdcd4 - by Bioz Stars, 2026-07
90/100 stars
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92
Boster Bio pdcd4
Expression of GAPLINC was detected in ox-LDL-induced cells. A, B – CCK-8 assay assessing cell proliferation ability in different times and concentrations of ox-LDL treatment conditions. C, D – Flow cytometry analysis of cell apoptosis rate in different times and concentrations of ox-LDL treatment conditions. * P < 0.05, ** p < 0.01, *** p < 0.001. E, F – Expression level of GAPLINC quantified by qPCR in different times and concentrations of ox-LDL treatment conditions. G, H – Relative expression level of miR-183-5p quantified by qPCR. I, J – Relative expression level of <t>PDCD4</t> quantified by qPCR. Mean ± SD were presented, and experiments were done in triplicate. * P < 0.05, ** p < 0.01, *** p < 0.001
Pdcd4, supplied by Boster Bio, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/cell+nuclear+antigen+inhibitor/pmc10895973-42-11-15?v=Boster+Bio
Average 92 stars, based on 1 article reviews
pdcd4 - by Bioz Stars, 2026-07
92/100 stars
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Image Search Results


Expression of GAPLINC was detected in ox-LDL-induced cells. A, B – CCK-8 assay assessing cell proliferation ability in different times and concentrations of ox-LDL treatment conditions. C, D – Flow cytometry analysis of cell apoptosis rate in different times and concentrations of ox-LDL treatment conditions. * P < 0.05, ** p < 0.01, *** p < 0.001. E, F – Expression level of GAPLINC quantified by qPCR in different times and concentrations of ox-LDL treatment conditions. G, H – Relative expression level of miR-183-5p quantified by qPCR. I, J – Relative expression level of PDCD4 quantified by qPCR. Mean ± SD were presented, and experiments were done in triplicate. * P < 0.05, ** p < 0.01, *** p < 0.001

Journal: Archives of Medical Science : AMS

Article Title: lncRNA GAPLINC regulates vascular endothelial cell apoptosis in atherosclerosis

doi: 10.5114/aoms/169383

Figure Lengend Snippet: Expression of GAPLINC was detected in ox-LDL-induced cells. A, B – CCK-8 assay assessing cell proliferation ability in different times and concentrations of ox-LDL treatment conditions. C, D – Flow cytometry analysis of cell apoptosis rate in different times and concentrations of ox-LDL treatment conditions. * P < 0.05, ** p < 0.01, *** p < 0.001. E, F – Expression level of GAPLINC quantified by qPCR in different times and concentrations of ox-LDL treatment conditions. G, H – Relative expression level of miR-183-5p quantified by qPCR. I, J – Relative expression level of PDCD4 quantified by qPCR. Mean ± SD were presented, and experiments were done in triplicate. * P < 0.05, ** p < 0.01, *** p < 0.001

Article Snippet: GAPDH (1 : 1000, Goodhere), Bcl2 (1 : 1000, Proteintech), and PDCD4 (1 : 2000, Boster) primary antibodies were incubated with the membrane overnight.

Techniques: Expressing, CCK-8 Assay, Flow Cytometry

GAPLINC interaction with miR-183-5p and PDCD4. The cells were treated with normal conditions, GAPLINC siRNA, GAPLINC overexpression, ox-LDL treatment, ox-LDL treatment with GAPLINC condition, ox-LDL treatment with GAPLINC siRNA and ox-LDL treatment with GAPLINC overexpression, respectively. A – A hypothesized model of GAPLINC downstream interaction molecules. B – Relative mRNA expression level of PDCD4, Bcl2, miR-183-5p and miR-211 * P < 0.05, ** p < 0.01, *** p < 0.001. C – Spearman correlation between miR-183-5p and PDCD4 was assessed. D – Relative protein expression levels of PDCD4 and Bcl2. Mean ± SD were presented, and experiments were done in triplicate. * P < 0.05, ** p < 0.01, *** p < 0.001

Journal: Archives of Medical Science : AMS

Article Title: lncRNA GAPLINC regulates vascular endothelial cell apoptosis in atherosclerosis

doi: 10.5114/aoms/169383

Figure Lengend Snippet: GAPLINC interaction with miR-183-5p and PDCD4. The cells were treated with normal conditions, GAPLINC siRNA, GAPLINC overexpression, ox-LDL treatment, ox-LDL treatment with GAPLINC condition, ox-LDL treatment with GAPLINC siRNA and ox-LDL treatment with GAPLINC overexpression, respectively. A – A hypothesized model of GAPLINC downstream interaction molecules. B – Relative mRNA expression level of PDCD4, Bcl2, miR-183-5p and miR-211 * P < 0.05, ** p < 0.01, *** p < 0.001. C – Spearman correlation between miR-183-5p and PDCD4 was assessed. D – Relative protein expression levels of PDCD4 and Bcl2. Mean ± SD were presented, and experiments were done in triplicate. * P < 0.05, ** p < 0.01, *** p < 0.001

Article Snippet: GAPDH (1 : 1000, Goodhere), Bcl2 (1 : 1000, Proteintech), and PDCD4 (1 : 2000, Boster) primary antibodies were incubated with the membrane overnight.

Techniques: Over Expression, Expressing

GAPLINC directly interacted with miR183-5p and modulated cell apoptosis. A – Luciferase reporter assay was performed to validate the direct target association of GAPLINC, miR-183 and PDCD4. The cells were treated with normal conditions, miR183-5p siRNA, miR183-5p overexpression, ox-LDL treatment, ox-LDL treatment with miR183-5p condition, ox-LDL treatment with miR183-5p siRNA and ox-LDL treatment with miR183-5p overexpression, respectively. B – Relative mRNA expression level of PDCD4 and miR-183-5p. C – CCK-8 assay assessing cell proliferation ability. * P < 0.05, ** p < 0.01, *** p < 0.001. D – Flow cytometry analysis of cell apoptosis rate. E – Cell apoptosis rate accessed by TUNEL assay. * P < 0.05, ** p < 0.01, *** p < 0.001. F – Cell cycle profiles were examined by flow cytometry. Grouped as G0/G1, G2/M and S stage; Mean ± SD were presented, and experiments were done in triplicate. * P < 0.05, ** p < 0.01, *** p < 0.001

Journal: Archives of Medical Science : AMS

Article Title: lncRNA GAPLINC regulates vascular endothelial cell apoptosis in atherosclerosis

doi: 10.5114/aoms/169383

Figure Lengend Snippet: GAPLINC directly interacted with miR183-5p and modulated cell apoptosis. A – Luciferase reporter assay was performed to validate the direct target association of GAPLINC, miR-183 and PDCD4. The cells were treated with normal conditions, miR183-5p siRNA, miR183-5p overexpression, ox-LDL treatment, ox-LDL treatment with miR183-5p condition, ox-LDL treatment with miR183-5p siRNA and ox-LDL treatment with miR183-5p overexpression, respectively. B – Relative mRNA expression level of PDCD4 and miR-183-5p. C – CCK-8 assay assessing cell proliferation ability. * P < 0.05, ** p < 0.01, *** p < 0.001. D – Flow cytometry analysis of cell apoptosis rate. E – Cell apoptosis rate accessed by TUNEL assay. * P < 0.05, ** p < 0.01, *** p < 0.001. F – Cell cycle profiles were examined by flow cytometry. Grouped as G0/G1, G2/M and S stage; Mean ± SD were presented, and experiments were done in triplicate. * P < 0.05, ** p < 0.01, *** p < 0.001

Article Snippet: GAPDH (1 : 1000, Goodhere), Bcl2 (1 : 1000, Proteintech), and PDCD4 (1 : 2000, Boster) primary antibodies were incubated with the membrane overnight.

Techniques: Luciferase, Reporter Assay, Over Expression, Expressing, CCK-8 Assay, Flow Cytometry, TUNEL Assay

Rescue experiments on GAPLINC. A – Relative expression level of GAPLINC, PDCD4 and miR-183-5p. Cells were treated with normal conditions, 50 μg of ox-LDL, 50 μg of ox-LDL-NC si-GAPLINC, si-miR-183-5p or both siRNA. B – CCK-8 assay assessing cell proliferation ability. * P < 0.05, ** p < 0.01, *** p < 0.001. C – Cell apoptosis rate accessed by TUNEL assay and flow cytometry analysis of cell apoptosis rate. D – Cell cycle profiles were examined by flow cytometry. E – Graphic model of mechanisms of GAPLINC. Mean ± SD were presented, and experiments were done in triplicate. * P < 0.05, ** p < 0.01, *** p < 0.001

Journal: Archives of Medical Science : AMS

Article Title: lncRNA GAPLINC regulates vascular endothelial cell apoptosis in atherosclerosis

doi: 10.5114/aoms/169383

Figure Lengend Snippet: Rescue experiments on GAPLINC. A – Relative expression level of GAPLINC, PDCD4 and miR-183-5p. Cells were treated with normal conditions, 50 μg of ox-LDL, 50 μg of ox-LDL-NC si-GAPLINC, si-miR-183-5p or both siRNA. B – CCK-8 assay assessing cell proliferation ability. * P < 0.05, ** p < 0.01, *** p < 0.001. C – Cell apoptosis rate accessed by TUNEL assay and flow cytometry analysis of cell apoptosis rate. D – Cell cycle profiles were examined by flow cytometry. E – Graphic model of mechanisms of GAPLINC. Mean ± SD were presented, and experiments were done in triplicate. * P < 0.05, ** p < 0.01, *** p < 0.001

Article Snippet: GAPDH (1 : 1000, Goodhere), Bcl2 (1 : 1000, Proteintech), and PDCD4 (1 : 2000, Boster) primary antibodies were incubated with the membrane overnight.

Techniques: Expressing, CCK-8 Assay, TUNEL Assay, Flow Cytometry